Trypanosoma cruzi induces edematogenic responses in mice and invades cardiomyocytes and endothelial cells in vitro by activating distinct kinin receptor subtypes (B1/B2)-Reference-Cited by-同舟云学术

Trypanosoma cruzi induces edematogenic responses in mice and invades cardiomyocytes and endothelial cells in vitro by activating distinct kinin receptor subtypes (B1/B2)

Published:2002-11 Issue:1 Volume:17 Page:73-75
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Todorov Alex G.¹,Andrade Daniele¹,Pesquero João B.²,Carvalho Araujo Ronaldo²,Bader Michael³,Stewart John⁴,Gera Lajos⁴,Müller‐Esterl Werner⁵,Morandi Verônica⁶,S. Goldenberg Regina C.³,Castro‐Faria Neto Hugo³⁷,Scharfstein Julio¹

Affiliation:

1. Instituto de Biofísica Carlos Chagas FilhoUniversidade do Brasil, Centro de Ciências da Saude Rio de Janeiro Brazil

2. Departmento de BiofísicaUniversidade Federal do Estado de São Paulo (UNIFESP), Escola Paulista de Medicina São Paulo Brazil

3. Max‐Delbruck‐Center for Molecular Medicine (MDC) Berlin‐Buch Germany

4. Department of Biochemistry and Molecular GeneticsUniversity of Colorado School of Medicine Denver Colorado 80262 USA

5. Institute for Biochemistry IIUniversity of Frankfurt Medical School Frankfurt Germany

6. Departamento de Biologia Celular e GenéticaUniversidade do Estado do Rio de Janeiro Rio de Janeiro Brazil

7. Instituto Oswaldo CruzFundação Oswaldo Cruz Rio de Janeiro Brazil

Funder

World Health Organization

Fundação Carlos Chagas Filho de Amparo à Pesquisa do Estado do Rio de Janeiro

Ministério da Ciência e Tecnologia

Fundação de Amparo à Pesquisa do Estado de São Paulo

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.02-0477fje

Reference36 articles.

1. Chagas' Disease

2. Immunity to Trypanosoma cruzi

3. Microvascular changes as a cause of chronic cardiomyopathy in Chagas' disease

4. Enhanced Platelet Adherence and Aggregation in Chagas' Disease: A Potential Pathogenic Mechanism for Cardiomyopathy

5. Immunopathology of Chagas disease

Cited by 83 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Interaction of Angiotensin-(1−7) with kinins in the kidney circulation: Role of B1 receptors;Peptides;2024-09

2. The ecotin‐like peptidase inhibitor of Trypanosoma cruzi prevents TMPRSS2‐PAR2‐TLR4 crosstalk downmodulating infection and inflammation;The FASEB Journal;2024-03-25

3. Creatine Supplementation Alleviates Fatigue after Exercise through Anti-Inflammatory Action in Skeletal Muscle and Brain;Nutraceuticals;2023-05-09

4. Genetic Ablation and Pharmacological Blockade of Bradykinin B1 Receptor Unveiled a Detrimental Role for the Kinin System in Chagas Disease Cardiomyopathy;Journal of Clinical Medicine;2023-04-15

5. Sheltered in Stromal Tissue Cells, Trypanosoma cruzi Orchestrates Inflammatory Neovascularization via Activation of the Mast Cell Chymase Pathway;Pathogens;2022-01-29