Prevention of cardiac fibrosis and left ventricular dysfunction in diabetic cardiomyopathy in rats by transgenic expression of the human tissue kallikrein gene

Author:

Tschöpe Carsten1,Walther Thomas1,Koniger Jens1,Spillmann Frank1,Westermann Dirk1,Escher Felicitas1,Pauschinger Matthias1,Pesquero Joao B.2,Bader Michael3,Schultheiss Heinz-Peter1,Noutsias Michel1

Affiliation:

1. Department of Cardiology and Pneumonology, Campus Benjamin FranklinCharité‐University MedicineFree University of BerlinBerlin

2. Department of BiophysicsUniversidade Federal de São PauloSão PauloBrazil

3. Max-Delbrück‐Center for Molecular MedicineBerlinGermany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

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1. A REVIEW ON THE CHEMICAL-INDUCED EXPERIMENTAL MODEL OF CARDIOTOXICITY;International Journal of Pharmacy and Pharmaceutical Sciences;2024-07-01

2. Bradykinin attenuates endothelial-mesenchymal transition following cardiac ischemia-reperfusion injury;European Journal of Pharmacology;2024-05

3. hKLK alleviates myocardial fibrosis in mice with viral myocarditis;Journal of Applied Biomedicine;2023-04-01

4. Mechanobiology of Cardiac Fibroblasts in Cardiac Remodeling;Cardiac Mechanobiology in Physiology and Disease;2023

5. Bibliometric analysis of the inflammation in diabetic cardiomyopathy;Frontiers in Cardiovascular Medicine;2022-12-13

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