Differential requirement for c‐Jun NH2‐terminal kinase in TNF‐α‐and Fas‐mediated apoptosis in hepatocytes-Reference-Cited by-同舟云学术

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Differential requirement for c‐Jun NH2‐terminal kinase in TNF‐α‐and Fas‐mediated apoptosis in hepatocytes

Published:2004-02-06 Issue:6 Volume:18 Page:720-722
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Schwabe Robert F.¹²,Uchinami Hiroshi¹²,Qian Ting³,Bennett Brydon L.⁴,Lemasters John J.³,Brenner David A.²

Affiliation:

1. Departments of MedicineUniversity of North CarolinaChapel HillNC

2. Biochemistry and BiophysicsUniversity of North CarolinaChapel HillNC

3. Cell and Developmental BiologyUniversity of North CarolinaChapel HillNC

4. Signal Research DivisionCelgene CorporationSan DiegoCA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.03-0771fje

Reference49 articles.

1. Signal Transduction by the JNK Group of MAP Kinases

2. AP-1 in cell proliferation and survival

3. Requirement of JNK for Stress- Induced Activation of the Cytochrome c-Mediated Death Pathway

4. The role of c‐Jun N‐terminal kinase (JNK) in apoptosis induced by ultraviolet C and gamma radiation. Duration of JNK activation may determine cell death and proliferation;Chen Y. R.;J. Biol. Chem.,1996

5. ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

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