Generation of complement component C5a by ischemic neurons promotes neuronal apoptosis-Reference-Cited by-同舟云学术

Generation of complement component C5a by ischemic neurons promotes neuronal apoptosis

Published:2012-05-31 Issue:9 Volume:26 Page:3680-3690
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Pavlovski Dale¹,Thundyil John¹,Monk Peter N.²,Wetsel Rick A.³,Taylor Stephen M.¹,Woodruff Trent M.¹

Affiliation:

1. School of Biomedical SciencesUniversity of QueenslandBrisbaneQueenslandAustralia

2. Department of Infection and ImmunityMedical SchoolUniversity of SheffieldSheffieldUK

3. Research Center for Immunology and Autoimmune DiseasesThe Brown Foundation Institute of Molecular Medicine for the Prevention of Human DiseasesUniversity of Texas Health Science Center at HoustonHoustonTexasUSA

Funder

Australian Research Council

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.11-202382

Reference50 articles.

1. Innate immunity in the nervous system;Ramaglia V.;Prog. Brain Res.,2009

2. Generation of C5a in the absence of C3: a new complement activation pathway

3. Complement component 5a (C5a)

4. Complement C3 and C5 play critical roles in traumatic brain cryoinjury: blocking effects on neutrophil extravasation by C5a receptor antagonist

5. Therapeutic activity of C5a receptor antagonists in a rat model of neurodegeneration

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