Adrenomedullary function is severely impaired in 21‐hydroxylase‐deficient mice-Reference-Cited by-同舟云学术

Adrenomedullary function is severely impaired in 21‐hydroxylase‐deficient mice

Published:1999-07 Issue:10 Volume:13 Page:1185-1194
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Hornstein S. R.¹,Tajima T.¹,Eisenhofer G.²,Haidan A.³,Aguilera G.¹

Affiliation:

1. Section on Endocrine PhysiologyDevelopmental Endocrinology BranchNational Institute of Child Health and Human Development Bethesda Maryland 20892‐1862 USA

2. National Institute of Neurological Disorders and StrokeNational Institutes of Health Bethesda Maryland 20892‐1862 USA

3. Department of Internal MedicineUniversity of Leipzig Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fasebj.13.10.1185

Reference71 articles.

1. Congenital adrenal hyperplasia due to 21‐hydroxylase deficiency;Cutler G. B.;N. Engl. J. Med.,1990

2. Diagnosis and Management of Congenital Adrenal Hyperplasia

3. Plasma Steroids in Congenital Adrenal Hyperplasia

4. ELEVATED ANDROGEN, BRAIN DEVELOPMENT AND LANGUAGE/LEARNING DISABILITIES IN CHILDREN WITH CONGENITAL ADRENAL HYPERPLASIA

5. Learning Disabilities in Children With Congenital Adrenal Hyperplasia

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