Defects in ryanodine receptor calcium release in skeletal muscle from postmyocardial infarcted rats-Reference-Cited by-同舟云学术

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Defects in ryanodine receptor calcium release in skeletal muscle from postmyocardial infarcted rats

Published:2003-06-03 Issue:11 Volume:17 Page:1-18
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Ward C. W.¹,Reiken S.²,Marks A. R.²,Marty I.³,Vassort G.⁴,Lacampagne A.⁴

Affiliation:

1. Department of Biochemistry and Molecular BiologyUniversity of Maryland School of Medicine Baltimore USA

2. Center for Molecular CardiologyColumbia University College of Physicians & Surgeons New York USA

3. INSERM E9931 DBMS /CEA Grenoble France

4. INSERM U390/IFR3Physiopathologie Cardiovasculaire CHU Arnaud de Villeneuve Montpellier France

Funder

Fondation pour la Recherche Médicale

Association Française contre les Myopathies

Hoechst Marion Roussel

Norges Idrettshøgskole

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.02-1083fje

Reference44 articles.

1. Lack of correlation between exercise capacity and indexes of resting left ventricular performance in heart failure

2. Exercise limitation in chronic heart failure: Central role of the periphery

3. HIGH TUMOUR NECROSIS FACTOR-α LEVELS ARE ASSOCIATED WITH EXERCISE INTOLERANCE AND NEUROHORMONAL ACTIVATION IN CHRONIC HEART FAILURE PATIENTS

4. Impaired skeletal muscle function in patients with congestive heart failure. Relationship to systemic exercise performance.

5. Predictors of exercise capacity in chronic heart failure

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5. Sphingomyelinase activity promotes atrophy and attenuates force in human muscle fibres and is elevated in heart failure patients;Journal of Cachexia, Sarcopenia and Muscle;2022-07-18

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