Implication of the MAGI‐1b/PTEN signalosome in stabilization of adherens junctions and suppression of invasiveness
Author:
Affiliation:
1. INSERM U410Faculté de Médecine X. BichatParis75018France
2. Department for Molecular Biomedical ResearchVIB‐Ghent UniversityB‐9052GhentBelgium
3. Laboratory of Experimental CancerologyGhent University HospitalB‐9000GhentBelgium
Funder
Institut National de la Santé et de la Recherche Médicale
Fonds De La Recherche Scientifique - FNRS
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.04-1942fje
Reference47 articles.
1. Genetic manipulation of E-cadherin expression by epithelial tumor cells reveals an invasion suppressor role
2. Cadherin Cell Adhesion Receptors as a Morphogenetic Regulator
3. MAGI-1 Interacts with β-Catenin and Is Associated with Cell–Cell Adhesion Structures
4. Inhibition by Platelet-activating Factor of Src- and Hepatocyte Growth Factor-dependent Invasiveness of Intestinal and Kidney Epithelial Cells
5. The lipid phosphatase activity of PTEN is critical for stabilizing intercellular junctions and reverting invasiveness
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