The neurovascular mechanism of clitoral erection: nitric oxide and cGMP‐stimulated activation of BKCachannels-Reference-Cited by-同舟云学术

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The neurovascular mechanism of clitoral erection: nitric oxide and cGMP‐stimulated activation of BKCachannels

Published:2004-09 Issue:12 Volume:18 Page:1382-1391
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Gragasin Ferrante S.¹,Michelakis Evangelos D.¹,Hogan Angie¹,Moudgil Rohit¹,Hashimoto Kyoko¹,Wu Xichen¹,Bonnet Sandra¹,Haromy Al¹,Archer Stephen L.¹

Affiliation:

1. Department of Medicine (Cardiology) and the Vascular Biology GroupUniversity of AlbertaEdmontonCanada

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.04-1978com

Reference57 articles.

1. SSI Prize Essay for Female Sexual Dysfunction—Clinical ‘Clinical evaluation of female sexual function: effects of age and estrogen status on subjective and physiologic sexual responses’

2. Experimental evidence for endothelium dependent relaxation and neuronal nitric oxide in corpus cavernosum

3. Nitric oxide and cyclic GMP formation upon electrical field stimulation cause relaxation of corpus cavernosum smooth muscle

4. Nitrergic neurotransmission mediates the non-adrenergic non-cholinergic responses in the clitoral corpus cavernosum of the rabbit

5. cGMP-dependent protein kinase activates Ca-activated K channels in cerebral artery smooth muscle cells

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