Abnormal maturation of the retinal vasculature in type XVIII collagen/endostatin deficient mice and changes in retinal glial cells due to lack of collagen types XV and XVIII-Reference-Cited by-同舟云学术

Abnormal maturation of the retinal vasculature in type XVIII collagen/endostatin deficient mice and changes in retinal glial cells due to lack of collagen types XV and XVIII

Published:2005-06-23 Issue:11 Volume:19 Page:1564-1566
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Hurskainen Merja¹,Eklund Lauri¹,Hägg Pasi O.²,Fruttiger Marcus³,Sormunen Raija⁴,Ilves Mika⁵,Pihlajaniemi Taina¹

Affiliation:

1. Collagen Research UnitBiocenter OuluDepartment of Medical Biochemistry and Molecular Biology Oulu Finland

2. Department of OphthalmologyUniversity of Oulu Oulu Finland

3. Wolfson Institute for Biomedical ResearchUniversity College London London United Kingdom

4. Biocenter OuluDepartment of PathologyUniversity of Oulu Oulu Finland

5. Biocenter OuluDepartment of PhysiologyUniversity of Oulu Oulu Finland

Funder

South Carolina Center of Economic Excellence

Academy of Finland

Sigrid Juséliuksen Säätiö

European Commission

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.04-3101fje

Reference46 articles.

1. Identification of Three N-terminal Ends of Type XVIII Collagen Chains and Tissue-specific Differences in the Expression of the Corresponding Transcripts

2. Collagen XVIII Is a Basement Membrane Heparan Sulfate Proteoglycan

3. Mouse Col18a1 is expressed in a tissue-specific manner as three alternative variants and is localized in basement membrane zones.

4. The Short and Long Forms of Type XVIII Collagen Show Clear Tissue Specificities in Their Expression and Location in Basement Membrane Zones in Humans

5. Lack of collagen XVIII/endostatin results in eye abnormalities

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