Monoamine oxidase‐A knockdown in human neuroblastoma cells reveals protection against mitochondrial toxins-Reference-Cited by-同舟云学术

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Monoamine oxidase‐A knockdown in human neuroblastoma cells reveals protection against mitochondrial toxins

Published:2013-09-19 Issue:1 Volume:28 Page:218-229
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Fitzgerald Julia C.¹²,Ugun‐Klusek Aslihan¹,Allen George³,De Girolamo Luigi A.¹,Hargreaves Iain⁴,Ufer Christoph¹⁵,Abramov Andrey Y.³,Billett E. Ellen¹

Affiliation:

1. School of Science and TechnologyNottingham Trent UniversityNottinghamUK

2. Functional Neurogenomics LaboratoryHertie Institute for Clinical Brain ResearchTübingenGermany

3. Department of Molecular NeuroscienceUniversity College London Institute of NeurologyLondonUK

4. Neurometabolic UnitNational Hospital for Neurology and NeurosurgeryLondonUK

5. Institute of BiochemistryUniversity Medicine Berlin‐CharitéBerlinGermany

Funder

Deutscher Akademischer Austauschdienst

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.13-235481

Reference60 articles.

1. Pro-apoptotic gene expression mediated by the p38 mitogen-activated protein kinase signal transduction pathway

2. Monoamine oxidase A and repressor R1 are involved in apoptotic signaling pathway

3. Monoamine oxidase-A modulates apoptotic cell death induced by staurosporine in human neuroblastoma cells

4. Serine 209 resides within a putative p38(MAPK) consensus motif and regulates monoamine oxidase-A activity

5. Mitochondria in neurodegenerative disorders: regulation of the redox state and death signaling leading to neuronal death and survival

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