FE65 interacts with ADP‐ribosylation factor 6 to promote neurite outgrowth
Author:
Affiliation:
1. School of Life SciencesChinese University of Hong KongShatinHong KongChina
2. King's College London Centre for Neurodegeneration ResearchDepartment of NeuroscienceKing's College London Institute of PsychiatryLondonUK
Funder
Wellcome Trust
Medical Research Council
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.13-232694
Reference91 articles.
1. The phosphotyrosine interaction domains of X11 and FE65 bind to distinct sites on the YENPTY motif of amyloid precursor protein
2. The Regions of the Fe65 Protein Homologous to the Phosphotyrosine Interaction/Phosphotyrosine Binding Domain of Shc Bind the Intracellular Domain of the Alzheimer's Amyloid Precursor Protein
3. The intracellular cytoplasmic domain of the Alzheimer's disease amyloid precursor protein interacts with phosphotyrosine-binding domain proteins in the yeast two-hybrid system
4. Phosphorylation-dependent Regulation of the Interaction of Amyloid Precursor Protein with Fe65 Affects the Production of β-Amyloid
5. Essential roles for the FE65 amyloid precursor protein-interacting proteins in brain development
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