FOXO3a mediates signaling crosstalk that coordinates ubiquitin and atrogin‐1/MAFbx expression during glucocorticoid‐induced skeletal muscle atrophy-Reference-Cited by-同舟云学术

FOXO3a mediates signaling crosstalk that coordinates ubiquitin and atrogin‐1/MAFbx expression during glucocorticoid‐induced skeletal muscle atrophy

Published:2010-04-06 Issue:8 Volume:24 Page:2660-2669
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Zheng Bin¹,Ohkawa Sakae¹,Li Haiyan¹,Roberts‐Wilson Tiffany K.²,Price S. Russ¹

Affiliation:

1. Renal DivisionEmory University School of MedicineEmory University Atlanta Georgia USA

2. Graduate Program in Biochemistry, Cell and Developmental Biology in the Graduate Division of Biological and Biomedical SciencesEmory University Atlanta Georgia USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.09-151480

Reference59 articles.

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2. Blockade of Glucocorticoid Receptor Binding and Inhibition of Dexamethasone-Induced Muscle Atrophy in the Rat by RU38486, a Potent Glucocorticoid Antagonist*

3. Acidosis and glucocorticoids concomitantly increase ubiquitin and proteasome subunit mRNAs in rat muscle

4. Glucocorticoids regulate mRNA levels for subunits of the 19 S regulatory complex of the 26 S proteasome in fast-twitch skeletal muscles

5. Activation of the ubiquitin pathway in rat skeletal muscle by catabolic doses of glucocorticoids

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