A self‐organized actomyosin drives multiple intercellular junction disruption and directly promotes neutrophil recruitment in lipopolysaccharide‐induced acute lung injury-Reference-Cited by-同舟云学术

A self‐organized actomyosin drives multiple intercellular junction disruption and directly promotes neutrophil recruitment in lipopolysaccharide‐induced acute lung injury

Published:2018-06-07 Issue:11 Volume:32 Page:6197-6211
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Chen Bing¹,Yang Zhen¹,Yang Congwen¹,Qin Wenhan¹,Gu Jianteng¹,Hu Chuanmin²,Chen An²,Ning Jiaolin¹,Yi Bin¹,Lu Kaizhi¹

Affiliation:

1. Department of AnesthesiaThird Military Medical UniversityChongqingChina

2. Department of Clinical Biochemistry, College of Medical Laboratory, Southwest HospitalThird Military Medical UniversityChongqingChina

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201701506RR

Reference53 articles.

1. Cytoskeletal mechanisms regulating vascular endothelial barrier function in response to acute lung injury

2. Outcomes and survival prediction models for severe adult acute respiratory distress syndrome treated with extracorporeal membrane oxygenation

3. Actin dynamics in the regulation of endothelial barrier functions and neutrophil recruitment during endotoxemia and sepsis

4. Sepsis and Endothelial Permeability

5. Endothelial dysfunction as a cellular mechanism for vascular failure

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