Effects of deficiency of Kelch‐like ECH‐associated protein 1 on skeletal organization: a mechanism for diminished nuclear factor of activated T cells cytoplasmic 1 during osteoclastogenesis-Reference-Cited by-同舟云学术

Effects of deficiency of Kelch‐like ECH‐associated protein 1 on skeletal organization: a mechanism for diminished nuclear factor of activated T cells cytoplasmic 1 during osteoclastogenesis

Published:2017-09 Issue:9 Volume:31 Page:4011-4022
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Sakai Eiko¹,Morita Masanobu²³,Ohuchi Masahiro³,Kido Mizuho A.⁴,Fukuma Yutaka⁵,Nishishita Kazuhisa¹,Okamoto Kuniaki¹,Itoh Ken⁶,Yamamoto Masayuki²,Tsukuba Takayuki¹

Affiliation:

1. Division of Dental PharmacologyDepartment of Developmental and Reconstructive MedicineNagasaki University Graduate School of Biomedical SciencesNagasakiJapan

2. Department of Medical BiochemistryTohoku University Graduate School of MedicineSendaiJapan

3. Department of Environmental Health Sciences and Molecular ToxicologyTohoku University Graduate School of MedicineSendaiJapan

4. Department of OrthodonticsGraduate School of Dental ScienceKyushu UniversityFukuokaJapan

5. Department of Anatomy and PhysiologyFaculty of MedicineSaga UniversitySagaJapan

6. Department of Stress Response ScienceHirosaki University Graduate School of MedicineHirosakiJapan

Funder

Japan Society for the Promotion of Science

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201700177R

Reference52 articles.

1. Birth and Death of Bone Cells: Basic Regulatory Mechanisms and Implications for the Pathogenesis and Treatment of Osteoporosis

2. Building strong bones: molecular regulation of the osteoblast lineage

3. Osteoclast differentiation and activation

4. Induction and Activation of the Transcription Factor NFATc1 (NFAT2) Integrate RANKL Signaling in Terminal Differentiation of Osteoclasts

5. NFATc1 Induces Osteoclast Fusion Via Up-Regulation of Atp6v0d2 and the Dendritic Cell-Specific Transmembrane Protein (DC-STAMP)

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