Genetic ablation of histone deacetylase 2 leads to lung cellular senescence and lymphoid follicle formation in COPD/emphysema-Reference-Cited by-同舟云学术

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Genetic ablation of histone deacetylase 2 leads to lung cellular senescence and lymphoid follicle formation in COPD/emphysema

Published:2018-04-09 Issue:9 Volume:32 Page:4955-4971
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Sundar Isaac K.¹,Rashid Kahkashan¹,Gerloff Janice¹,Rangel-Moreno Javier²,Li Dongmei³,Rahman Irfan¹

Affiliation:

1. Department of Environmental MedicineUniversity of Rochester Medical CenterRochesterNew YorkUSA

2. Division of Allergy, Immunology, and Rheumatology, Department of MedicineUniversity of Rochester Medical CenterRochesterNew YorkUSA

3. Department of Clinical and Translational ResearchUniversity of Rochester Medical CenterRochesterNew YorkUSA

Funder

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201701518R

Reference70 articles.

1. Role of histone deacetylase 2 in epigenetics and cellular senescence: implications in lung inflammaging and COPD

2. Histone deacetylases in replicative senescence: evidence for a senescence-specific form of HDAC-2

3. Human HDAC1 and HDAC2 function in the DNA-damage response to promote DNA nonhomologous end-joining

4. Histone deacetylase 2–mediated deacetylation of the glucocorticoid receptor enables NF-κB suppression

5. Nrf2 deficiency influences susceptibility to steroid resistance via HDAC2 reduction

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