Hydroxysteroid (17β) dehydrogenase 1 expressed by Sertoli cells contributes to steroid synthesis and is required for male fertility-Reference-Cited by-同舟云学术

Hydroxysteroid (17β) dehydrogenase 1 expressed by Sertoli cells contributes to steroid synthesis and is required for male fertility

Published:2018-01-24 Issue:6 Volume:32 Page:3229-3241
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Hakkarainen Janne¹,Zhang Fu-Ping¹²,Jokela Heli¹,Mayerhofer Artur³,Behr Rüdiger⁴⁵,Cisneros-Montalvo Sheyla¹,Nurmio Mirja¹⁶,Toppari Jorma¹⁶,Ohlsson Claes⁷,Kotaja Noora¹,Sipilä Petra¹,Poutanen Matti¹²⁷

Affiliation:

1. Institute of BiomedicineResearch Centre for Integrative Physiology and PharmacologyUniversity of Turku Turku Finland

2. Turku Center for Disease ModelingUniversity of Turku Turku Finland

3. Cell Biology–Anatomy IIIBiomedical Center (BMC)Ludwig-Maximilians-Universität München Martinsried Germany

4. Platform Degenerative DiseasesGerman Primate Center, Leibniz Institute for Primate Research Göttingen Germany

5. German Center for Cardiovascular Research (DZHK)Partner Site Göttingen Göttingen Germany

6. Department of PediatricsTurku University Hospital Turku Finland

7. Institute of Medicinethe Sahlgrenska AcademyGothenburg University Gothenburg Sweden

Funder

Academy of Finland

Sigrid Juséliuksen Säätiö

Vetenskapsrådet

IngaBritt och Arne Lundbergs Forskningsstiftelse

Sahlgrenska Universitetssjukhuset

Novo Nordisk Fonden

Deutscher Akademischer Austauschdienst

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201700921R

Reference55 articles.

1. Mouse Spermatogenic Stem Cells Continually Interconvert between Equipotent Singly Isolated and Syncytial States

2. FSH and testosterone signaling in Sertoli cells

3. Endocrine Regulation of Spermatogenesis

4. Complete Sertoli Cell Proliferation Induced by Follicle-Stimulating Hormone (FSH) Independently of Luteinizing Hormone Activity: Evidence from Genetic Models of Isolated FSH Action

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