Ischemic preconditioning modulates the expression of several genes, leading to the overproduction of IL‐1Ra, iNOS, and Bcl‐2 in a human model of liver ischemia‐reperfusion

Author:

Barrier Alain123,Olaya Natalia1,Chiappini Franck1,Roser François1,Scatton Olivier14,Artus Cédric1,Franc Brigitte5,Dudoit Sandrine2,Flahault Antoine3,Debuire Brigitte1,Azoulay Daniel14,Lemoine Antoinette1

Affiliation:

1. Inserm 602Service de Biochimie et Biologie MoléculaireHôpital Universitaire Paul BrousseUniversité Paris‐Sud/XIAssistance Publique‐Hôpitaux de Paris Villejuif Cedex France

2. Division of BiostatisticsUniversity of California Berkeley USA

3. Inserm U444Université Paris‐VIHôpital Tenon Paris

4. Centre Hépato‐BiliaireHôpital Paul Brousse Villejuif

5. Service d'Anatomie PathologieHôpital Ambroise Paré Boulogne‐Billancourt France

Funder

Novartis

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference68 articles.

1. Studies of necrosis in vitro of mouse hepatic parenchymal cells. Ultrastructural alterations in endoplasmic reticulum, Golgi apparatus, plasma membrane, and lipid droplets;Trump B. F.;Lab. Invest.,1965

2. Mitochondrial permeability transition in pH-dependent reperfusion injury to rat hepatocytes

3. Morphologic, biochemical, and molecular evidence of apoptosis during the reperfusion phase after brief periods of renal ischemia;Schumer M.;Am. J. Pathol.,1992

4. Reperfusion injury induces apoptosis in rabbit cardiomyocytes;Gottlieb R. A.;J. Clin. Invest.,1994

5. Apoptosis after ischemia‐reperfusion in human liver allografts;Borghi‐Scoazec G.;Liver Transpl. Surg.,1997

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