CK2 activity is required for the interaction of FGF14 with voltage‐gated sodium channels and neuronal excitability-Reference-Cited by-同舟云学术

CK2 activity is required for the interaction of FGF14 with voltage‐gated sodium channels and neuronal excitability

Published:2016-02-25 Issue:6 Volume:30 Page:2171-2186
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Hsu Wei‐Chun J.¹,Scala Federico¹²,Nenov Miroslav N.¹,Wildburger Norelle C.¹³,Elferink Hannah⁴,Singh Aditya K.¹,Chesson Charles B.⁵,Buzhdygan Tetyana¹,Sohail Maveen⁶,Shavkunov Alexander S.¹,Panova Neli I.¹,Nilsson Carol L.¹⁷,Rudra Jai S.¹,Lichti Cheryl F.¹⁸,Laezza Fernanda¹⁸⁹¹⁰

Affiliation:

1. Department of Pharmacology and Toxicology

2. Institute of Human Physiology, Università CattolicaRomeItaly

3. Department of NeurologyWashington University School of MedicineSt. LouisMissouriUSA

4. Center in Environmental Toxicology

5. Human Pathophysiology and Translational MedicineInstitute for Translational SciencesUniversity of Texas Medical BranchGalvestonTexasUSA

6. Graduate School for Biomedical SciencesUniversity of Texas Medical BranchGalvestonTexasUSA

7. Sealy Center for Molecular MedicineUniversity of Texas Medical BranchGalvestonTexasUSA

8. Mitchell Center for Neurodegenerative DiseasesUniversity of Texas Medical BranchGalvestonTexasUSA

9. Center for Addiction ResearchUniversity of Texas Medical BranchGalvestonTexasUSA

10. Center for Biomedical EngineeringUniversity of Texas Medical BranchGalvestonTexasUSA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201500161

Reference51 articles.

1. Fibroblast Growth Factor Homologous Factors Control Neuronal Excitability through Modulation of Voltage-Gated Sodium Channels

2. Preferential Targeting of Nav1.6 Voltage-Gated Na+ Channels to the Axon Initial Segment during Development

3. Fibroblast Growth Factor Homologous Factor 2B: Association with Nav1.6 and Selective Colocalization at Nodes of Ranvier of Dorsal Root Axons

4. The FGF14F145S Mutation Disrupts the Interaction of FGF14 with Voltage-Gated Na+ Channels and Impairs Neuronal Excitability

5. FGF14 N-terminal splice variants differentially modulate Nav1.2 and Nav1.6-encoded sodium channels

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