Chronic stress accelerates learning and memory impairments and increases amyloid deposition in APP V717I ‐CT100 transgenic mice, an Alzheimer's disease model-Reference-Cited by-同舟云学术

Chronic stress accelerates learning and memory impairments and increases amyloid deposition in APP V717I ‐CT100 transgenic mice, an Alzheimer's disease model

Published:2006-02-08 Issue:6 Volume:20 Page:729-731
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Jeong Yun Ha¹,Park Cheol Hyoung¹,Yoo Jongman¹,Shin Ki Young¹,Ahn Sung‐Min¹,Kim Hye‐Sun¹,Lee Sang Hyung²,Emson Piers C.³,Suh Yoo‐Hun¹

Affiliation:

1. Department of PharmacologyCollege of Medicine, National Creative Research Initiative Centre for Alzheimer's Dementia and Neuroscience Research Institute, MRC, Seoul National University Seoul Korea

2. Department of NeurosurgeryCollege of MedicineSeoul National University Seoul Korea

3. Neurobiology ProgrammeThe Babraham Institute Babraham Cambridge United Kingdom

Funder

Ministry of Education, Science and Technology

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.05-4265fje

Reference65 articles.

1. Mutant presenilins of Alzheimer's disease increase production of 42residue amyloid beta‐protein in both transfected cells and transgenic mice;Selkoe D. J.;Nat. Med.,1997