Genetic or nutritional disorders in homocysteine or folate metabolism increase proteinN‐homocysteinylation in mice-Reference-Cited by-同舟云学术

Genetic or nutritional disorders in homocysteine or folate metabolism increase proteinN‐homocysteinylation in mice

Published:2009-02-09 Issue:6 Volume:23 Page:1721-1727
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Jakubowski Hieronim¹²,Perła-Kaján Joanna¹²,Finnell Richard H.¹³,Cabrera Robert M.³,Wang Hong⁴,Gupta Sapna⁵,Kruger Warren D.⁵,Kraus Jan P.⁶,Shih Diana M.⁷

Affiliation:

1. Department of Microbiology and Molecular GeneticsNew Jersey Medical SchoolUniversity of Medicine and Dentistry of New JerseyInternational Center for Public HealthNewarkNew JerseyUSA

2. Department of Biochemistry and BiotechnologyUniversity of Life SciencesPoznaPoland

3. Texas Institute for Genomic MedicineInstitute of Biosciences and TechnologyThe Texas A&M University System Health Science CenterHoustonTexasUSA

4. Department of PharmacologyTemple University School of MedicinePhiladelphiaPennsylvaniaUSA

5. Division of PopulationFox Chase Cancer CenterPhiladelphiaPennsylvaniaUSA

6. Department of PediatricsUniversity of Colorado School of MedicineAuroraColoradoUSA

7. Department of MedicineUniversity of CaliforniaLos Angeles Medical SchoolLos AngelesCaliforniaUSA

Funder

American Heart Association

Ministerstwo Nauki i Szkolnictwa Wyższego

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.08-127548

Reference43 articles.

1. Synthesis of homocysteine thiolactone by methionyl-tRNA synthetase in cultured mammalian cells

2. Translational Accuracy of Aminoacyl-tRNA Synthetases: Implications for Atherosclerosis

3. Molecular basis of homocysteine toxicity in humans

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