Ahnak is critical for cardiac Ca(v)1.2 calcium channel function and its β‐adrenergic regulation-Reference-Cited by-同舟云学术

Ahnak is critical for cardiac Ca(v)1.2 calcium channel function and its β‐adrenergic regulation

Published:2005-12 Issue:14 Volume:19 Page:1969-1977
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Haase Hannelore¹,Alvarez Julio²,Petzhold Daria¹,Doller Anke³,Behlke Joachim¹,Erdmann Jeanette⁴,Hetzer Roland⁵,Regitz‐Zagrosek Vera³⁵,Vassort Guy⁶,Morano Ingo¹⁷

Affiliation:

1. Max Delbrück Center for Molecular Medicine (MDC) D‐13092 Berlin Germany

2. Institute of Cardiology La Habana Cuba

3. Center for Cardiovascular ResearchUniversity Medicine Charité Berlin Germany

4. Medizinische Klinik IIUniversity UK‐SH Campus Lübeck Germany

5. Deutsches Herzzentrum Berlin Germany

6. Physiopathologie CardiovasculaireINSERM U‐637 Montpellier Cedex 5 France

7. Johannes Müller Institute for PhysiologyUniversity Medicine Charite Berlin Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.05-3997com

Reference41 articles.

1. Ionic basis of ventricular arrhythmias in remodeled rat heart during long-term myocardial infarction

2. Molecular Basis of Electrical Remodeling in Atrial Fibrillation

3. Cardiac-specific Overexpression of the α1 Subunit of the L-type Voltage-dependent Ca2+ Channel in Transgenic Mice

4. Increased Availability and Open Probability of Single L-Type Calcium Channels From Failing Compared With Nonfailing Human Ventricle

5. Structure and Regulation of Voltage-Gated Ca2+ Channels

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