Heme oxygenase‐1‐derived carbon monoxide protects hearts from transplant‐associated ischemia reperfusion injury-Reference-Cited by-同舟云学术

Heme oxygenase‐1‐derived carbon monoxide protects hearts from transplant‐associated ischemia reperfusion injury

Published:2004-02-20 Issue:6 Volume:18 Page:771-772
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Akamatsu Yorihiro¹,Haga Manabu¹,Tyagi Shivraj¹,Yamashita Kenichiro¹,Graça‐Souza Aurelio Vicente¹,Ollinger Robert¹,Czismadia Eva¹,May G. Aaron²,Ifedigbo Emeka²,Otterbein Leo E.²,Bach Fritz H.¹,Soares Miguel P.¹³

Affiliation:

1. Immunobiology Research CenterDepartment of SurgeryBeth Israel Deaconess Medical CenterHarvard Medical SchoolBostonMA02215

2. University of Pittsburgh School of MedicinePittsburghPA

3. Instituto Gulbenkian de CiênciaApartado 142781–901OeirasPortugal

Funder

National Institutes of Health

Pfizer

American Heart Association

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.03-0921fje

Reference53 articles.

1. Tolerance, Danger, and the Extended Family

2. COMPLEMENT DEPOSITION IN EARLY CARDIAC TRANSPLANT BIOPSIES IS ASSOCIATED WITH ISCHEMIC INJURY AND SUBSEQUENT REJECTION EPISODES1

3. Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement.

4. The neutrophil as a mediator of myocardial ischemia-reperfusion injury: time to move on

5. Oxidative stress and neutrophil activation—the two keystones of ischemia/reperfusion injury

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