The pleiotropic mouse phenotype extra‐toes spotting is caused by translation initiation factorEif3cmutations and is associated with disrupted sonic hedgehog signaling-Reference-Cited by-同舟云学术

The pleiotropic mouse phenotype extra‐toes spotting is caused by translation initiation factorEif3cmutations and is associated with disrupted sonic hedgehog signaling

Published:2011-02-03 Issue:5 Volume:25 Page:1596-1605
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Gildea Derek E.¹²,Luetkemeier Erin S.²,Bao Xiaozhong³,Loftus Stacie K.²,Mackem Susan³,Yang Yingzi²,Pavan William J.²,Biesecker Leslie G.²

Affiliation:

1. Institute for Biomedical SciencesGeorge Washington UniversityWashingtonDistrict of ColumbiaUSA

2. National Human Genome Research InstituteNational Institutes of HealthBethesdaMarylandUSA

3. National Cancer InstituteNational Institutes of HealthBethesdaMarylandUSA

Funder

National Human Genome Research Institute

National Cancer Institute

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.10-169771

Reference37 articles.

1. Extra-toes spotting, a newpolydactylymutation;Lyon M.;Mouse News Letter,1989

2. Ko V.(1998)A genomics approach to the study of mouse limb development: mapping of CA150 and the genetics of extra‐toes spotting.Undergraduate thesis Princeton University Princeton NJ USA

3. A mouse model of Greig cephalo–polysyndactyly syndrome: the extra–toesJ mutation contains an intragenic deletion of the Gli3 gene

4. High-resolution mapping of the Gli3 mutation Extra-toesJ reveals a 51.5-kb deletion

5. Deletion of GLI3 supports the homology of the human Greig cephalopolysyndactyly syndrome (GCPS) and the mouse mutant extra toes (Xt)

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