Specific residues of the cytoplasmic domains of cardiac inward rectifier potassium channels are effective antifibrillatory targets-Reference-Cited by-同舟云学术

Specific residues of the cytoplasmic domains of cardiac inward rectifier potassium channels are effective antifibrillatory targets

Published:2010-06-28 Issue:11 Volume:24 Page:4302-4312
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Noujaim Sami F.¹,Stuckey Jeanne A.²,Ponce‐Balbuena Daniela³,Ferrer‐Villada Tania³,López‐Izquierdo Angelica³,Pandit Sandeep¹,Calvo Conrado J.¹,Grzeda Krzysztof R.¹,Berenfeld Omer¹,Sánchez Chapula José A.³,Jalife José¹

Affiliation:

1. Center for Arrhythmia Research Ann Arbor Michigan USA

2. Life Sciences InstituteUniversity of Michigan Ann Arbor Michigan USA

3. Unidad de Investigación Carlos Méndez del Centro Universitario de Investigaciones Biomedicas de la Universidad de Colima Colima México

Funder

National Heart, Lung, and Blood Institute

Fondation Leducq

American Heart Association

University of Michigan Center for Gastrointestinal Research

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.10-163246

Reference42 articles.

1. Inwardly Rectifying Potassium Channels: Their Structure, Function, and Physiological Roles

2. A Novel Form of Short QT Syndrome (SQT3) Is Caused by a Mutation in the KCNJ2 Gene

3. A Kir2.1 gain-of-function mutation underlies familial atrial fibrillation

4. Human inward rectifier potassium channels in chronic and postoperative atrial fibrillation

5. Report of the NASPE/NHLBI Round Table on Future Research Directions in Atrial Fibrillation

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