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Alcohol consumption is controlled by angiotensin II

Published:2001-05-29 Issue:9 Volume:15 Page:1640-1642
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Maul Bjoern¹,Siems Wolf-Eberhard¹,Hoehe Margret R.²,Grecksch Gisela³,Bader Michael⁴,Walther Thomas⁴⁵

Affiliation:

1. Institute of Molecular Pharmacology Berlin Germany

2. Genome Research, Max-Delbrueck-Center for Molecular Medicine (MDC) Berlin‐Buch Germany

3. Institute of Pharmacology and ToxicologyUniversity of Magdeburg Magdeburg Germany

4. Hypertension ResearchMax-Delbrueck-Center for Molecular Medicine (MDC) Berlin‐Buch Germany

5. Department of Cardiology and PneumologyUniversity HospitalBenjamin Franklin Free University of Berlin Germany

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.00-0797fje

Reference29 articles.

1. Elevated alcohol consumption in null mutant mice lacking 5-HT1B serotonin receptors;Crabbe J. C.;Nat. Genet.,1996

2. Alcohol preference and sensitivity are markedly reduced in mice lacking dopamine D2 receptors

3. Ethanol consumption and resistance are inversely related to neuropeptide Y levels

4. Angiotensin and captopril increase alcohol intake

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5. The effect of telmisartan, an angiotensin receptor blocker, on alcohol consumption and alcohol-induced dopamine release in the nucleus accumbens;Alcohol;2021-11

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