Peroxiredoxin‐2 recycling is slower in denser and pediatric sickle cell red cells-Reference-Cited by-同舟云学术

Peroxiredoxin‐2 recycling is slower in denser and pediatric sickle cell red cells

Published:2022-03-20 Issue:4 Volume:36 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Oh Joo‐Yeun¹²,Bae Chae Yun¹²,Kasztan Malgorzata³,Pollock David M.⁴,Russell Robert T.⁵,Lebensburger Jeffrey³,Patel Rakesh P.¹²^ORCID

Affiliation:

1. Department of Pathology University of Alabama at Birmingham Birmingham Alabama USA

2. Center for Free Radical Biology University of Alabama at Birmingham Birmingham Alabama USA

3. Pediatric Hematology Oncology University of Alabama at Birmingham Birmingham Alabama USA

4. Section of Cardio‐Renal Physiology and Medicine Division of Nephrology Department of Medicine University of Alabama at Birmingham Birmingham Alabama USA

5. Division of Pediatric Surgery Department of Surgery, Children's of Alabama University of Alabama at Birmingham Birmingham Alabama USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202200052R

Reference35 articles.

1. Irreversible Inactivation of Glutathione Peroxidase 1 and Reversible Inactivation of Peroxiredoxin II by H2O2in Red Blood Cells

2. The effects of disruption of genes for peroxiredoxin-2, glutathione peroxidase-1, and catalase on erythrocyte oxidative metabolism

3. Peroxiredoxin II is essential for sustaining life span of erythrocytes in mice

4. Peroxiredoxin 2 functions as a noncatalytic scavenger of low-level hydrogen peroxide in the erythrocyte

5. Red blood cell oxidative stress impairs oxygen delivery and induces red blood cell aging

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