Tafazzin deficiency in mouse mesenchymal stem cells promote reprogramming of activated B lymphocytes toward immunosuppressive phenotypes-Reference-Cited by-同舟云学术

Tafazzin deficiency in mouse mesenchymal stem cells promote reprogramming of activated B lymphocytes toward immunosuppressive phenotypes

Published:2022-07-11 Issue:8 Volume:36 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Zegallai Hana M.¹,Abu‐El‐Rub Ejlal²³,Olayinka‐Adefemi Folayemi⁴,Cole Laura K.¹,Sparagna Genevieve C.⁵,Marshall Aaron J.⁴,Hatch Grant M.¹^ORCID

Affiliation:

1. Diabetes Research Envisioned and Accomplished in Manitoba (DREAM) Theme, Children's Hospital Research Institute of Manitoba, Department of Pharmacology & Therapeutics, Rady Faculty of Health Sciences University of Manitoba Winnipeg Manitoba Canada

2. Physiology and Pathophysiology, Department of Basic Medical Sciences, Faculty of Medicine Yarmouk University Irbid Jordan

3. Physiology and Pathophysiology, Regenerative Medicine, Faculty of Medicine University of Manitoba Winnipeg Manitoba Canada

4. Department of Immunology, Rady Faculty of Health Sciences University of Manitoba Winnipeg Manitoba Canada

5. Department of Medicine, Division of Cardiology University of Colorado Anschutz Medical Center Aurora Colorado USA

Funder

Canada Research Chairs

Institute of Infection and Immunity

Natural Sciences and Engineering Research Council of Canada

Children's Hospital Research Institute of Manitoba

University of Manitoba

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202200145R

Reference62 articles.

1. Cardiolipin metabolism and Barth Syndrome

2. Barth syndrome

3. The Enzymatic Function of Tafazzin

4. Making heads or tails of phospholipids in mitochondria

5. Barth syndrome: cardiolipin, cellular pathophysiology, management, and novel therapeutic targets

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