The fetal origins of atherosclerosis: maternal hypercholesterolemia, and cholesterol‐lowering or antioxidant treatment during pregnancy influence in utero programming and postnatal susceptibility to atherogenesis-Reference-Cited by-同舟云学术

The fetal origins of atherosclerosis: maternal hypercholesterolemia, and cholesterol‐lowering or antioxidant treatment during pregnancy influence in utero programming and postnatal susceptibility to atherogenesis

Published:2002-09 Issue:11 Volume:16 Page:1348-1360
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

PALINSKI WULF¹,NAPOLI CLAUDIO²

Affiliation:

1. Department of Medicine 0682University of California San Diego La Jolla California USA

2. Department of MedicineFederico II University of Naples 80131 Naples Italy

Funder

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.02-0226rev

Reference90 articles.

1. Cellular pathology of progressive atherosclerosis in the WHHL rabbit. An animal model of familial hypercholesterolemia.

2. Histological findings and evidence of lipid conjugated dienes and malonyldialdehyde in human fetal aortas;D'Armiento F. P.;Acta Paediatr.,1993

3. Fatty streak formation occurs in human fetal aortas and is greatly enhanced by maternal hypercholesterolemia. Intimal accumulation of low density lipoprotein and its oxidation precede monocyte recruitment into early atherosclerotic lesions.

4. Intracranial Arteries of Human Fetuses Are More Resistant to Hypercholesterolemia-Induced Fatty Streak Formation Than Extracranial Arteries

5. Pathophysiological Events during Pregnancy Influence the Development of Atherosclerosis in Humans

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