CuZnSOD gene deletion targeted to skeletal muscle leads to loss of contractile force but does not cause muscle atrophy in adult mice-Reference-Cited by-同舟云学术

CuZnSOD gene deletion targeted to skeletal muscle leads to loss of contractile force but does not cause muscle atrophy in adult mice

Published:2013-05-31 Issue:9 Volume:27 Page:3536-3548
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Zhang Yiqiang¹²,Davis Carol¹,Sakellariou George K.³,Shi Yun⁴²,Kayani Anna C.³,Pulliam Daniel⁴,Bhattacharya Arunabh⁴²,Richardson Arlan⁴²⁵,Jackson Malcolm J.³,McArdle Anne³,Brooks Susan V.⁶,Van Remmen Holly⁴²⁵

Affiliation:

1. Department of PhysiologyUniversity of Texas Health Science CenterSan AntonioTexasUSA

2. Barshop Institute for Longevity and Aging StudiesUniversity of Texas Health Science CenterSan AntonioTexasUSA

3. Department of Musculoskeletal BiologyInstitute of Ageing and Chronic DiseaseUniversity of LiverpoolLiverpoolUK

4. Department of Cellular and Structural BiologyUniversity of Texas Health Science CenterSan AntonioTexasUSA

5. Geriatric Research, Education, and Clinical Center (GRECC)South Texas Veterans Health SystemSan AntonioTexasUSA

6. Department of Molecular and Integrative PhysiologyUniversity of MichiganAnn ArborMichiganUSA

Funder

National Institute on Aging

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.13-228130

Reference37 articles.

1. Review Article: Sarcopenia: Causes, Consequences, and Preventions

2. From animals to humans: evidence linking oxidative stress as a causative factor in muscle atrophy

3. Role of superoxide-nitric oxide interactions in the accelerated age-related loss of muscle mass in mice lacking Cu,Zn superoxide dismutase

4. Skeletal muscle weakness due to deficiency of CuZn-superoxide dismutase is associated with loss of functional innervation

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