RBM20 phosphorylation and its role in nucleocytoplasmic transport and cardiac pathogenesis-Reference-Cited by-同舟云学术

RBM20 phosphorylation and its role in nucleocytoplasmic transport and cardiac pathogenesis

Published:2022-04-08 Issue:5 Volume:36 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Zhang Yanghai¹,Wang Chunyan¹,Sun Mingming²,Jin Yutong³,Braz Camila Urbano¹,Khatib Hasan¹,Hacker Timothy A.⁴,Liss Martin⁵,Gotthardt Michael⁵⁶,Granzier Henk⁷,Ge Ying³⁸⁹,Guo Wei¹²^ORCID

Affiliation:

1. Department of Animal and Dairy Sciences University of Wisconsin‐Madison Wisconsin USA

2. Department of Animal Science University of Wyoming Laramie Wyoming USA

3. Department of Chemistry University of Wisconsin‐Madison Madison Wisconsin USA

4. Division of Cardiovascular Medicine Department of Medicine University of Wisconsin School of Medicine and Public Health Madison Wisconsin USA

5. Neuromuscular and Cardiovascular Cell Biology Max Delbrueck Center for Molecular Medicine in the Helmholtz Association Berlin Germany

6. Charité Universitätsmedizin Berlin Germany

7. Department of Cellular and Molecular Medicine University of Arizona Tucson Arizona USA

8. Department of Cell and Regenerative Biology School of Medicine and Public Health University of Wisconsin‐Madison Madison Wisconsin USA

9. Human Proteomics Program School of Medicine and Public Health University of Wisconsin‐Madison Madison Wisconsin USA

Funder

National Institute of General Medical Sciences

American Heart Association

Wisconsin Alumni Research Foundation

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202101811RR

Reference65 articles.

1. Mutation that dramatically alters rat titin isoform expression and cardiomyocyte passive tension

2. RBM20, a gene for hereditary cardiomyopathy, regulates titin splicing

3. Pathophysiological Defects and Transcriptional Profiling in the RBM20-/- Rat Model

4. Rbm20 regulates titin alternative splicing as a splicing repressor

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