CD226 deficiency promotes glutaminolysis and alleviates mitochondria damage in vascular endothelial cells under hemorrhagic shock-Reference-Cited by-同舟云学术

CD226 deficiency promotes glutaminolysis and alleviates mitochondria damage in vascular endothelial cells under hemorrhagic shock

Published:2021-10-20 Issue:11 Volume:35 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Zhou Shangxun¹²,Xie Jiangang¹²,Yu Chaoping¹²,Feng Zhusheng²,Cheng Kun¹,Ma Jingchang¹,Wang Yuling¹,Duan Chujun²,Zhang Yuan³,Jin Boquan¹,Yin Wen²,Zhuang Ran¹³^ORCID

Affiliation:

1. Department of Immunology Fourth Military Medical University Xi'an China

2. Department of Emergency Xijing Hospital Fourth Military Medical University Xi'an China

3. Institute of Medical Research Northwestern Polytechnical University Xi'an China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202101134R

Reference63 articles.

1. Hemorrhagic Shock

2. A whiter shade of pale: the ongoing challenge of haemorrhagic shock;Muckart DJJ;Anaesthesiol Intensive Ther,2018

3. Loss of Syndecan-1 Abrogates the Pulmonary Protective Phenotype Induced by Plasma After Hemorrhagic Shock

4. Evolving functions of endothelial cells in inflammation

5. Inflammation, glucose, and vascular cell damage: the role of the pentose phosphate pathway

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