GRIM‐19 is a target of mycobacterial Zn 2+ metalloprotease 1 and indispensable for NLRP3 inflammasome activation-Reference-Cited by-同舟云学术

GRIM‐19 is a target of mycobacterial Zn 2+ metalloprotease 1 and indispensable for NLRP3 inflammasome activation

Published:2021-12-14 Issue:1 Volume:36 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Kurane Tomomi¹,Matsunaga Tetsuro²,Ida Tomoaki²,Sawada Kazuko³,Nishimura Akira²,Fukui Masayuki³,Umemura Masayuki¹³⁴,Nakayama Masaaki⁵,Ohara Naoya⁵,Matsumoto Sohkichi⁶,Akaike Takaaki²,Matsuzaki Goro¹³⁴^ORCID,Takaesu Giichi¹³⁴^ORCID

Affiliation:

1. Department of Host Defense, Graduate School of Medicine University of the Ryukyus Okinawa Japan

2. Department of Environmental Medicine and Molecular Toxicology Tohoku University Graduate School of Medicine Sendai Japan

3. Molecular Microbiology Group, Tropical Biosphere Research Center University of the Ryukyus Okinawa Japan

4. Advanced Medical Research Center, Faculty of Medicine University of the Ryukyus Okinawa Japan

5. Department of Oral Microbiology, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences Okayama University Okayama Japan

6. Department of Bacteriology Niigata University School of Medicine Niigata Japan

Funder

Takeda Science Foundation

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202101074RR

Reference40 articles.

1. Prevention of phagosome-lysosome fusion in cultured macrophages by sulfatides of Mycobacterium tuberculosis.

2. M. tuberculosis and M. leprae Translocate from the Phagolysosome to the Cytosol in Myeloid Cells

3. ESX-1 dependent impairment of autophagic flux byMycobacterium tuberculosisin human dendritic cells

4. Mycobacterium tuberculosis Prevents Inflammasome Activation

5. IL-1β Promotes Antimicrobial Immunity in Macrophages by Regulating TNFR Signaling and Caspase-3 Activation

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