GORAB promotes embryonic lung maturation through antagonizing AKT phosphorylation, versican expression, and mesenchymal cell migration-Reference-Cited by-同舟云学术

GORAB promotes embryonic lung maturation through antagonizing AKT phosphorylation, versican expression, and mesenchymal cell migration

Published:2020-02-18 Issue:4 Volume:34 Page:4918-4933
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Liu Ying¹²³,Chen Xi⁴⁵,Choi Yeon Ja⁴,Yang Ning⁴,Song Zhongya⁴⁶,Snedecor Elizabeth R.⁴,Liang Wei¹²³,Leung Elaine Lai‐Han⁵,Zhang Lianfeng¹²³,Qin Chuan¹²³,Chen Jiang¹²³⁴⁷

Affiliation:

1. Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences Ministry of Health Beijing China

2. Comparative Medical Center, Peking Union Medical College Ministry of Health Beijing China

3. Key Laboratory of Human Disease Comparative Medicine Ministry of Health Beijing China

4. Department of Pathology Stony Brook University Stony Brook NY USA

5. State Key Laboratory of Quality Research in Chinese Medicine Macau University of Science and Technology Macau China

6. Department of Dermatology Peking University First Hospital Beijing China

7. Department of Dermatology Stony Brook University Stony Brook NY USA

Funder

Stony Brook University

China Scholarship Council

Science and Technology Development Fund

National Natural Science Foundation of China

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201902075R

Reference64 articles.

1. Gerodermia osteodysplastica is caused by mutations in SCYL1BP1, a Rab-6 interacting golgin

2. GORAB Missense Mutations Disrupt RAB6 and ARF5 Binding and Golgi Targeting

3. Gorab Is Required for Dermal Condensate Cells to Respond to Hedgehog Signals during Hair Follicle Morphogenesis

4. Impaired proteoglycan glycosylation, elevated TGF-β signaling, and abnormal osteoblast differentiation as the basis for bone fragility in a mouse model for gerodermia osteodysplastica

5. SCYL1BP1 modulates neurite outgrowth and regeneration by regulating the Mdm2/p53 pathway