Ceruloplasmin deficiency does not induce macrophagic iron overload: lessons from a new rat model of hereditary aceruloplasminemia-Reference-Cited by-同舟云学术

Ceruloplasmin deficiency does not induce macrophagic iron overload: lessons from a new rat model of hereditary aceruloplasminemia

Published:2019-12 Issue:12 Volume:33 Page:13492-13502
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Kenawi Moussa¹,Rouger Emmanuel¹,Island Marie-Laure¹,Leroyer Patricia¹,Robin François¹,Rémy Séverine²,Tesson Laurent²,Anegon Ignacio²,Nay Kévin³,Derbré Frédéric³,Brissot Pierre¹,Ropert Martine¹,Cavey Thibault¹,Loréal Olivier¹

Affiliation:

1. INSERM, Univ Rennes, INRA, Platform Analyse Elémentaire et Métabolisme des Métaux, UMR 1241 Institut NuMeCan (Nutrition, Metabolism, and Cancer), Centre Hospitalier Universitaire (CHU) Pontchaillou, Rennes, France;

2. INSERM UMR 1064- Centre de Recherches en Transplantation et Immunologie (CRTI), Transgenic Rats ImmunoPhenomic facility, Nantes, France;

3. Laboratory Movement, Sport, and Health Sciences (M2S-EA7470), University Rennes 2–Ecole Normale Supérieure (ENS) Rennes, Bruz, France

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference66 articles.

1. Aceruloplasminemia

2. Characterization, mapping, and expression of the human ceruloplasmin gene.

3. Anemia and iron overload due to compound heterozygosity for novel ceruloplasmin mutations

4. Aceruloplasminemia: new clinical, pathophysiological and therapeutic insights

5. Hepatic but not brain iron is rapidly chelated by deferasirox in aceruloplasminemia due to a novel gene mutation

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