Activity‐dependent cleavage of dyskinesia‐related proline‐rich transmembrane protein 2 (PRRT2) by calpain in mouse primary cortical neurons
Author:
Affiliation:
1. Department of Genome‐based Drug Discovery Graduate School of Biomedical Sciences Nagasaki University Nagasaki‐shi Japan
2. Department of Clinical Psychiatry Graduate School of Medicine Kagawa University Kita‐gun Japan
Funder
Japan Society for the Promotion of Science
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201902148R
Reference50 articles.
1. Exome sequencing identifies truncating mutations in PRRT2 that cause paroxysmal kinesigenic dyskinesia
2. Mutations in PRRT2 responsible for paroxysmal kinesigenic dyskinesias also cause benign familial infantile convulsions
3. PRRT2 Mutations Cause Benign Familial Infantile Epilepsy and Infantile Convulsions with Choreoathetosis Syndrome
4. PRRT2 mutations in familial infantile seizures, paroxysmal dyskinesia, and hemiplegic migraine
5. A Novel Topology of Proline-rich Transmembrane Protein 2 (PRRT2)
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3. Proline-rich transmembrane protein 2 knock-in mice present dopamine-dependent motor deficits;The Journal of Biochemistry;2023-10-04
4. Clinical and genetic analysis of benign familial infantile epilepsy caused by PRRT2 gene variant;Frontiers in Neurology;2023-05-09
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