The HDAC3 inhibitor RGFP966 ameliorated ischemic brain damage by downregulating the AIM2 inflammasome-Reference-Cited by-同舟云学术

The HDAC3 inhibitor RGFP966 ameliorated ischemic brain damage by downregulating the AIM2 inflammasome

Published:2019-11-26 Issue:1 Volume:34 Page:648-662
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Zhang Mei‐Juan¹,Zhao Qiu‐Chen¹²,Xia Ming‐Xu¹,Chen Jian¹,Chen Yan‐Ting¹,Cao Xiang¹,Liu Yi¹,Yuan Zeng‐Qiang³,Wang Xiao‐Ying²,Xu Yun¹⁴⁵

Affiliation:

1. Department of Neurology Drum Tower Hospital Medical School and The State Key Laboratory of Pharmaceutical Biotechnology Nanjing University Nanjing China

2. Departments of Neurology Massachusetts General Hospital Harvard Medical School Charlestown MA USA

3. Institute of Basic Medical Sciences Academy of Military Medical Sciences Beijing China

4. Jiangsu Key Laboratory for Molecular Medicine Medical School of Nanjing University Nanjing China

5. Jiangsu Province Stroke Center for Diagnosis and Therapy Nanjing China

Funder

Meijuan Zhang

Yun Xu

Yanting Chen

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201900394RRR

Reference49 articles.

1. Histone deacetylase (HDAC) inhibitors targeting HDAC3 and HDAC1 ameliorate polyglutamine-elicited phenotypes in model systems of Huntington's disease

2. Tubastatin A, an HDAC6 inhibitor, alleviates stroke-induced brain infarction and functional deficits: potential roles of α-tubulin acetylation and FGF-21 up-regulation

3. Requirement for the histone deacetylase Hdac3 for the inflammatory gene expression program in macrophages

4. Proteomic Analysis of HDAC3 Selective Inhibitor in the Regulation of Inflammatory Response of Primary Microglia

5. Expression patterns of histone deacetylases in experimental stroke and potential targets for neuroprotection

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