EB1‐ and EB2‐dependent anterograde trafficking of TRPM4 regulates focal adhesion turnover and cell invasion-Reference-Cited by-同舟云学术

EB1‐ and EB2‐dependent anterograde trafficking of TRPM4 regulates focal adhesion turnover and cell invasion

Published:2019-05-21 Issue:8 Volume:33 Page:9434-9452
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Blanco Constanza¹²,Morales Danna³²,Mogollones Ignacio¹²,Vergara‐Jaque Ariela³⁴⁵,Vargas Carla¹²,Álvarez Alhejandra¹²,Riquelme Denise⁶,Leiva‐Salcedo Elías⁶,González Wendy²⁵,Morales Diego¹²,Maureira Diego¹²,Aldunate Ismael¹,Cáceres Mónica¹²⁷,Varela Diego³²,Cerda Oscar¹²⁷

Affiliation:

1. Program of Cellular and Molecular Biology Universidad de Chile Santiago Chile

2. Millennium Nucleus of Ion Channels‐Associated Diseases (MiNICAD) Santiago Chile

3. Program of Physiology and Biophysics Institute of Biomedical Sciences Faculty of Medicine Universidad de Chile Santiago Chile

4. Multidisciplinary Scientific Nucleus Universidad de Talca Talca Chile

5. Center for Bioinformatics and Molecular Simulation Universidad de Talca Talca Chile

6. Department of Biology Faculty of Chemistry and Biology Universidad de Santiago de Chile Santiago Chile

7. The Wound Repair Treatment, and Health (WoRTH) Initiative Santiago Chile

Funder

Fondo Nacional de Desarrollo Científico y Tecnológico

Ministerio de Educación, Gobierno de Chile

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201900136R

Reference91 articles.

1. TRPM4 Is a Ca2+-Activated Nonselective Cation Channel Mediating Cell Membrane Depolarization

2. The Ca2+-activated cation channel TRPM4 is regulated by phosphatidylinositol 4,5-biphosphate

3. Regulation of the Ca2+ Sensitivity of the Nonselective Cation Channel TRPM4

4. The calcium-activated nonselective cation channel TRPM4 is essential for the migration but not the maturation of dendritic cells

5. TRPM4 controls insulin secretion in pancreatic β-cells

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