Complexes formed with integrin‐α5 and KCNB1 potassium channel wild type or epilepsy‐susceptibility variants modulate cellular plasticity via Ras and Akt signaling-Reference-Cited by-同舟云学术

Complexes formed with integrin‐α5 and KCNB1 potassium channel wild type or epilepsy‐susceptibility variants modulate cellular plasticity via Ras and Akt signaling

Published:2019-11-02 Issue:12 Volume:33 Page:14680-14689
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Yu Wei¹,Shin Mi Ryung¹,Sesti Federico¹

Affiliation:

1. Department of Neuroscience and Cell BiologyRobert Wood Johnson Medical School, Rutgers UniversityPiscatawayNew JerseyUSA

Funder

National Science Foundation

National Institutes of Health

National Institute on Aging

National Institute of Neurological Disorders and Stroke

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201901792R

Reference71 articles.

1. Non-conducting functions of voltage-gated ion channels

2. New Insights into the Regulation of Ion Channels by Integrins

3. Oxidation of KCNB1 channels in the human brain and in mouse model of Alzheimer’s disease

4. Oxidation of KCNB1 potassium channels triggers apoptotic integrin signaling in the brain

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