Inflammasome-mediated innate immunity in Alzheimer’s disease-Reference-Cited by-同舟云学术

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Inflammasome-mediated innate immunity in Alzheimer’s disease

Published:2019-12 Issue:12 Volume:33 Page:13075-13084
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Venegas Carmen¹,Heneka Michael T.¹²³

Affiliation:

1. Department of Neurodegenerative Disease and Geriatric Psychiatry, University of Bonn Medical Center, Bonn, Germany;

2. German Center for Neurodegenerative Disease (DZNE), Bonn, Germany; and

3. Department of Infectious Diseases and Immunology, University of Massachussetts Medical School, Worcester, Massachusetts, USA

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference104 articles.

1. NLRs join TLRs as innate sensors of pathogens

2. The inflammasomes: mechanisms of activation and function

3. Inflammasomes: too big to miss

4. Danger-associated molecular patterns in Alzheimer’s disease

5. Cryopyrin/NALP3 binds ATP/dATP, is an ATPase, and requires ATP binding to mediate inflammatory signaling

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1. A Review of the Retinal Impact of Traumatic Brain Injury and Alzheimer’s Disease: Exploring Inflammasome Complexes and Nerve Fiber Layer Alterations;Cureus;2024-08-17

2. Inflammasome links traumatic brain injury, chronic traumatic encephalopathy, and Alzheimer’s disease;Neural Regeneration Research;2024-07-10

3. The neuroinflammatory role of microglia in Alzheimer's disease and their associated therapeutic targets;CNS Neuroscience & Therapeutics;2024-07

4. Chronic kidney disease leads to microglial potassium efflux and inflammasome activation in the brain;Kidney International;2024-07

5. Pyroptosis of Vascular Smooth Muscle Cells as a Potential New Target for Preventing Vascular Diseases;Cardiovascular Drugs and Therapy;2024-06-01

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