The VPAC 2 agonist peptide histidine isoleucine (PHI) up‐regulates glutamate transport in the corpus callosum of a rat model of amyotrophic lateral sclerosis (hSOD1 G93A ) by inhibiting caspase‐3 mediated inactivation of GLT‐1a-Reference-Cited by-同舟云学术

The VPAC 2 agonist peptide histidine isoleucine (PHI) up‐regulates glutamate transport in the corpus callosum of a rat model of amyotrophic lateral sclerosis (hSOD1 G93A ) by inhibiting caspase‐3 mediated inactivation of GLT‐1a

Published:2011-07-05 Issue:10 Volume:25 Page:3674-3686
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Goursaud Stéphanie¹,Focant Marylène C.¹,Berger Julie V.¹,Nizet Yannick²,Maloteaux Jean‐Marie¹,Hermans Emmanuel¹

Affiliation:

1. Group of Neuropharmacology, Institute of NeuroscienceUniversité Catholique de Louvain Brussels Belgium

2. Laboratory of Experimental SurgeryInstitute of Experimental and Clinical Research, Université Catholique de Louvain Brussels Belgium

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.11-182337

Reference59 articles.

1. Corpus callosum involvement is a consistent feature of amyotrophic lateral sclerosis

2. Diffusion tensor MRI of early upper motor neuron involvement in amyotrophic lateral sclerosis

3. Correlation between pyramidal tract degeneration and widespread white matter involvement in amyotrophic lateral sclerosis: A study with tractography and diffusion-tensor imaging

4. Biomarkers in amyotrophic lateral sclerosis

5. Callosal dysfunction in amyotrophic lateral sclerosis correlates with diffusion tensor imaging of the central motor system

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