The mitochondrial permeability transition initiates autophagy in rat hepatocytes

Author:

Elmore Steven P.1,Qian Ting2,Grissom Sherry F.2,Lemasters John J.2

Affiliation:

1. Laboratory for PhysiologyVrije UniversityVan der Boechorststraat 71081 BTAmsterdamThe Netherlands

2. Department of CellDevelopmental BiologyCurriculum in ToxicologyUniversity of North CarolinaChapel HillNorth Carolina

Funder

Center for Gastrointestinal Biology and Disease, School of Medicine, University of North Carolina at Chapel Hill

National Institutes of Health

National Institute of Environmental Health Sciences

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference48 articles.

1. Studies on cellular autophagocytosis. A histochemical study on sequential alterations of mitochondria in the glucagon‐induced autophagic vacuoles of rat liver;Arstila A.;Lab. Invest.,1972

2. Amino acid and hormonal control of macromolecular turnover in perfused rat liver. Evidence for selective autophagy;Lardeux B. R.;J. Biol. Chem.,1987

3. Response of autophagic protein degradation to physiologic and pathologic stimuli in rat hepatocyte monolayer cultures;Yu Q. C.;Lab. Invest.,1988

4. Quantitative relationship between autophagy and proteolysis during graded amino acid deprivation in perfused rat liver;Schworer C. M.;J. Biol. Chem.,1981

5. Amino acid control of autophagic sequestration and protein degradation in isolated rat hepatocytes.

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