Soluble glycoprotein VI dimer inhibits platelet adhesion and aggregation to the injured vessel wall in vivo-Reference-Cited by-同舟云学术

Soluble glycoprotein VI dimer inhibits platelet adhesion and aggregation to the injured vessel wall in vivo

Published:2003-12-04 Issue:2 Volume:18 Page:1-18
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Massberg Steffen¹,Konrad Ildiko¹,Bültmann Andreas²,Schulz Christian¹,Münch Götz²,Peluso Mario²,Lorenz Michael¹,Schneider Simon¹,Besta Felicitas¹,Müller Iris¹,Hu Bin¹,Langer Harald¹,Kremmer Elisabeth³,Rudelius Martina⁴,Heinzmann Ulrich⁵,Ungerer Martin²,Gawaz Meinrad¹

Affiliation:

1. Klinikum rechts der Isar, 1. Medizinische Klinik Technische Universität München Ismaningerstrasse 22 D‐81675 München Germany

2. Procorde GmbH Martinsried, Fraunhoferstrasse 9 D‐82152 München Germany

3. Institute of Molecular Immunology GSF‐National Research Center for Environment and Health Marchionini Strasse 25 D‐81377 München Germany

4. Institut für Pathologie, Klinikum rechts der Isar Technische Universität München Ismaningerstrasse 22 D‐80567 München Germany

5. GSF National Research Center for Environment and Health Institute of Pathology Ingolstädter Landstraße 1 D‐85764 Neuherberg Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.03-0464fje

Reference31 articles.

1. Platelet interaction with collagen fibrils in flowing blood. I. Reaction of human platelets with alpha chymotrypsin‐digested subendothelium;Baumgartner H. R.;Thromb. Haemost.,1977

2. Platelet Collagen Receptors

3. A Crucial Role of Glycoprotein VI for Platelet Recruitment to the Injured Arterial Wall In Vivo

4. A patient with platelets deficient in glycoprotein VI that lack both collagen-induced aggregation and adhesion.

5. The Platelet Collagen Receptor Glycoprotein VI Is a Member of the Immunoglobulin Superfamily Closely Related to FcαR and the Natural Killer Receptors

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