Nucleotide excision repair DNA synthesis by excess DNA polymerase β: a potential source of genetic instability in cancer cells-Reference-Cited by-同舟云学术

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Nucleotide excision repair DNA synthesis by excess DNA polymerase β: a potential source of genetic instability in cancer cells

Published:2000-09 Issue:12 Volume:14 Page:1765-1774
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Canitrot Yvan¹,Hoffmann Jean-Sébastien¹,Calsou Patrick²,Hayakawa Hiroshi³,Salles Bernard³,Cazaux Christophe¹

Affiliation:

1. Groupe ‘Instabilité génétique et cancer’CNRS UMR 5089, 31077 Toulouse cedex 4 France

2. Groupe ‘Toxico‐resistance’, Institut de Pharmacologie et de Biologie StructuraleCNRS UMR 5089, 31077 Toulouse cedex 4 France

3. Department of BiochemistryMedical school of Kyushu UniversityFukuoka 812-8582 Japan

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.99-1063com

Reference64 articles.

1. DNA synthesis, mismatch repair, and cancer;Hoffmann J. S.;Int. J. Oncol.,1997

2. Genetic instabilities in human cancers

3. Characterization of DNA polymerase β mRNA: cell-cycle and growth response in cultured human cells

4. Induction of beta-polymerase mRNA by DNA-damaging agents in Chinese hamster ovary cells.

5. Frameshift mutagenesis by eukaryotic DNA polymerases in vitro;Kunkel T. A.;J. Biol. Chem.,1986

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