A role for the TIM‐3/GAL‐9/BAT3 pathway in determining the clinical phenotype of multiple sclerosis
Author:
Affiliation:
1. Don C. Gnocchi FoundationIstituto di Ricovero e Cura a Carattere Scientifico (IRCCS)MilanItaly
2. Department of Physiopathology and TransplantsUniversity of MilanMilanItaly
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.14-258194
Reference44 articles.
1. TIM-3 is expressed on activated human CD4+ T cells and regulates Th1 and Th17 cytokines
2. TIM-1 and TIM-3 enhancement of Th2 cytokine production by mast cells
3. Promotion of Tissue Inflammation by the Immune Receptor Tim-3 Expressed on Innate Immune Cells
4. T Cell Ig- and Mucin-Domain-Containing Molecule-3 (TIM-3) and TIM-1 Molecules Are Differentially Expressed on Human Th1 and Th2 Cells and in Cerebrospinal Fluid-Derived Mononuclear Cells in Multiple Sclerosis
5. T-bet, a Th1 transcription factor regulates the expression of Tim-3
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