Causative role of oxidative stress in a Drosophila model of Friedreich ataxia-Reference-Cited by-同舟云学术

Causative role of oxidative stress in a Drosophila model of Friedreich ataxia

Published:2006-12-13 Issue:2 Volume:21 Page:333-344
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Llorens José V.¹,Navarro Juan A.²,Martínez‐Sebastián Maria J.¹,Baylies Mary K.³,Schneuwly S.²,Botella José A.²,Moltó Maria D.¹

Affiliation:

1. Departament de Genètica Universitat de València Burjassot Valencia Spain

2. Institute of Zoology University of Regensburg Regensburg Germany

3. Developmental Biology Program Memorial Sloan Kettering Cancer Institute New York New York USA

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.05-5709com

Reference68 articles.

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2. Clinical features and classification of inherited ataxias;Harding A. E.;Adv. Neurol,1993

3. The heart disease of Friedreich's ataxia: a clinical and electrocardiographic study of 115 patients, with an analysis of serial electrocardiographic changes in 30 cases;Harding A. E.;Q. J. Med,1983

4. Friedreich's Ataxia: Autosomal Recessive Disease Caused by an Intronic GAA Triplet Repeat Expansion

5. Sticky DNA, a Self-associated Complex Formed at Long GAA·TTC Repeats in Intron 1 of the Frataxin Gene, Inhibits Transcription

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