The selective cyclooxygenase‐2 inhibitor SC‐236 reduces liver fibrosis by mechanisms involving non‐parenchymal cell apoptosis and PPARγ activation-Reference-Cited by-同舟云学术

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The selective cyclooxygenase‐2 inhibitor SC‐236 reduces liver fibrosis by mechanisms involving non‐parenchymal cell apoptosis and PPARγ activation

Published:2005-05-04 Issue:9 Volume:19 Page:1120-1122
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Planagumà Anna¹,Clària Joan¹,Miquel Rosa²,López‐Parra Marta¹,Titos Esther¹,Masferrer Jaime L.¹,Arroyo Vicente³,Rodé Joan³

Affiliation:

1. DNA UnitHospital ClínicInstitut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS)Universitat de Barcelona Barcelona Spain

2. Pathology LaboratoryHospital ClínicInstitut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS)Universitat de Barcelona Barcelona Spain

3. Liver UnitHospital ClínicInstitut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS)Universitat de Barcelona Barcelona Spain

Funder

Ministerio de Ciencia y Tecnología

Instituto de Salud Carlos III

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.04-2753fje

Reference53 articles.

1. Mechanisms of Disease: mechanisms of hepatic fibrosis and therapeutic implications

2. New aspects of hepatic fibrosis

3. Current management of alcoholic liver disease

4. Inhibition by acetylsalicylic acid, a cyclo-oxygenase inhibitor, and p-bromophenacylbromide, a phospholipase A2 inhibitor, of both cirrhosis and enzyme-altered nodules caused by a choline-deficient, L-amino acid-defined diet in rats

5. Prostaglandins and other cyclooxygenase-dependent arachidonic acid metabolites and the kidney in liver disease

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