Reactive oxygen species alter brain endothelial tight junction dynamics via RhoA, PI3 kinase, and PKB signaling-Reference-Cited by-同舟云学术

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Reactive oxygen species alter brain endothelial tight junction dynamics via RhoA, PI3 kinase, and PKB signaling

Published:2007-06-22 Issue:13 Volume:21 Page:3666-3676
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Schreibelt Gerty¹,Kooij Gijs¹,Reijerkerk Arie¹,Doorn Ruben¹,Gringhuis Sonja I.¹,Pol Susanne¹,Weksler Babette B.²,Romero Ignacio A.³,Couraud Pierre‐Olivier⁴,Piontek Jorg⁵,Blasig Ingolf E.⁵,Dijkstra Christine D.¹,Ronken Eric⁶,Vries Helga E.¹

Affiliation:

1. Department of Molecular Cell Biology and ImmunologyVU University Medical CenterAmsterdamThe Netherlands

2. Weill Medical CollegeCornell UniversityNew YorkUSA

3. Department of Biological SciencesThe Open UniversityWalton HallMilton KeynesUK

4. Institut CochinDepartement de Biologie CellulaireParisFrance

5. Institute of Molecular PharmacologyBerlinGermany

6. Solvay Pharmaceuticals Research LaboratoriesWeespThe Netherlands

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.07-8329com

Reference62 articles.

1. The Blood-Brain Barrier and Its Role in Immune Privilege in the Central Nervous System

2. Occludin: a novel integral membrane protein localizing at tight junctions.

3. Occludin and claudins in tight-junction strands: leading or supporting players?

4. Multiple domains of occludin are involved in the regulation of paracellular permeability

5. Claudin-1 and -2: Novel Integral Membrane Proteins Localizing at Tight Junctions with No Sequence Similarity to Occludin

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