Mitochondria‐targeted antioxidants protect Friedreich Ataxia fibroblasts from endogenous oxidative stress more effectively than untargeted antioxidants-Reference-Cited by-同舟云学术

Mitochondria‐targeted antioxidants protect Friedreich Ataxia fibroblasts from endogenous oxidative stress more effectively than untargeted antioxidants

Published:2003-08-15 Issue:13 Volume:17 Page:1-10
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Jauslin Matthias L.¹,Meier Thomas¹,Smith Robin A. J.²,Murphy P. Michael³

Affiliation:

1. MyoContract Ltd. CH-4410 Liestal Switzerland

2. Chemistry DepartmentUniversity of Otago Dunedin New Zealand

3. MRC-Dunn Human Nutrition Unit Cambridge CB2 2XY United Kingdom

Funder

European Commission

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.03-0240fje

Reference21 articles.

1. Friedreich's Ataxia: Autosomal Recessive Disease Caused by an Intronic GAA Triplet Repeat Expansion

2. Friedreich's ataxia is a mitochondrial disorder

3. Deficit of in vivo mitochondrial ATP production in patients with Friedreich ataxia

4. Recent advances in the molecular pathogenesis of Friedreich ataxia

5. Mouse models for Friedreich ataxia exhibit cardiomyopathy, sensory nerve defect and Fe-S enzyme deficiency followed by intramitochondrial iron deposits

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