Glucose clearance and uptake is increased in the SOD1 G93A mouse model of amyotrophic lateral sclerosis through an insulin‐independent mechanism
Author:
Affiliation:
1. School of Biomedical Sciences The University of Queensland, St Lucia Brisbane QLD Australia
2. Queensland Brain Institute The University of Queensland, St Lucia Brisbane QLD Australia
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202002450R
Reference48 articles.
1. Amyotrophic Lateral Sclerosis
2. Skeletal Muscle in Motor Neuron Diseases: Therapeutic Target and Delivery Route for Potential Treatments
3. Clinical Spectrum of Amyotrophic Lateral Sclerosis (ALS)
4. A comprehensive review of amyotrophic lateral sclerosis
5. Metabolic Dysfunctions in Amyotrophic Lateral Sclerosis Pathogenesis and Potential Metabolic Treatments
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