Inhibition of ACSL4 attenuates ferroptotic damage after pulmonary ischemia‐reperfusion-Reference-Cited by-同舟云学术

Inhibition of ACSL4 attenuates ferroptotic damage after pulmonary ischemia‐reperfusion

Published:2020-10-18 Issue:12 Volume:34 Page:16262-16275
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Xu Yixin¹²,Li Xuehan¹²,Cheng Yan²,Yang Mingan³,Wang Rurong¹

Affiliation:

1. Department of Anesthesiology West China HospitalSichuan UniversityThe Research Units of West China (2018RU12)Chinese Academy of Medical Sciences Chengdu China

2. Laboratory of Anesthesia & Critical Care Medicine Translational Neuroscience CenterWest China HospitalSichuan University Chengdu China

3. Division of Biostatistics & Epidemiology School of Public HealthSan Diego State University San Diego CA USA

Funder

Department of Science and Technology of Sichuan Province

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202001758R

Reference43 articles.

1. Primary graft dysfunction

2. Mechanisms of lung ischemia-reperfusion injury

3. Inhibition of regulated necrosis attenuates receptor-interacting protein kinase 1–mediated ischemia-reperfusion injury after lung transplantation

4. Autophagy induced by DAMPs facilitates the inflammation response in lungs undergoing ischemia-reperfusion injury through promoting TRAF6 ubiquitination

5. Tau-mediated iron export prevents ferroptotic damage after ischemic stroke

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